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Benclamet (Glibenclamide)

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Benclamet belongs to a class of drugs known as sulfonylureas. These drugs act by closing ATP-sensitive potassium channels on pancreatic beta cells. The ATP-sensitive potassium channels on beta cells are known as sulfonylurea receptor 1 (SUR1).

Other names for this medication:
Aviglen, Benclamet, Benformin, Betanase, Betrol, Combidos, Comdiab, Daocon, Diabetnil, Diabetrol, Diabnil, Diolin, Duotrol, Euglucon, Giclamide, Glibact, Glibenclamide, Glibet, Glibson, Glime, Glimsug, Glinil, Glitrac, Glucomin, Glucosafe, Glucosil, Glucotrac, Glunil, Glutowin, Glybenclamide, Glybonil, Glyboral, Glybovin, Glybovine, Glycurb, Glycurub, Glymep, Iscept, Melitus, Metclam, Metica, Pionorm, Sugatrol, Sugnil, Sugnl, Sugronil, Symet, Triglucored, Triglycomet

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Norvasc, Lozol, Pepcid, Buspar, Zithromax, Motilium, Maxolon, Remeron, Elavil, Lamictal, Roical, Dumocycline, Talendep, Syntac, Eriacta, Depsonil,

Also known as: Glibenclamide


This medication is sometimes prescribed for other uses; ask your doctor or pharmacist for more information.


Usual Adult Dose for Diabetes Type 2:

Doses provided as Benclamet-metformin -As Initial Therapy in Patients with Inadequate Glycemic Control on Diet and Exercise Alone: Starting dose: 1.25 mg-250 mg orally once a day -Consider a starting dose of 1.25 mg-250 mg orally twice a day for patients with fasting blood glucose (FBG) greater than 200 mg/dL or HbA1c greater than 9% Maintenance Dose: Increase in increments of 1.25 mg-250 mg per day every 2 weeks up to the minimum effective dose to achieve glycemic control Maximum Initial Dose: 10 mg-2000 mg per day -Patients with Inadequate Glycemic Control on a Benclamet (or another Sulfonylurea) and/or Metformin: Initial dose: 2.5 mg-500 mg or 5 mg-500 mg orally twice a day Maintenance Dose: Increase in increments of no more than 5 mg-500 mg to the minimum effective dose to achieve adequate blood glucose control Maximum Dose: 20 mg-2000 mg per day Comments: -Give with meals; 5 mg-500 mg dose should not be used as initial therapy due to an increased risk of hypoglycemia; initial doses should be conservative to avoid hypoglycemia largely due to Benclamet and gastrointestinal side effects largely due to metformin. -For patients who are switching to combination therapy, initial doses should not exceed the daily dose of Benclamet (or equivalent sulfonylurea) and metformin already being taken; the decision to switch to the nearest equivalent dose should be based on clinical judgement. -Addition of thiazolidinedione may be appropriate for patients not adequately controlled on Benclamet-metformin; in patients experiencing hypoglycemia, consider reducing the dose of the Benclamet component. -Monitor patients closely for signs and symptoms of hypoglycemia. Use: As an adjunct to diet and exercise to improve glycemic control in patients with type 2 diabetes mellitus.

Detailed Benclamet/metformin dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. An overdose can cause severe hypoglycemia or lactic acidosis.


Keep this medication in the container it came in, tightly closed, and out of reach of children. Store it at room temperature and away from excess heat and moisture (not in the bathroom).

It is important to keep all medication out of sight and reach of children as many containers (such as weekly pill minders and those for eye drops, creams, patches, and inhalers) are not child-resistant and young children can open them easily. To protect young children from poisoning, always lock safety caps and immediately place the medication in a safe location – one that is up and away and out of their sight and reach.

Unneeded medications should be disposed of in special ways to ensure that pets, children, and other people cannot consume them. However, you should not flush this medication down the toilet. Instead, the best way to dispose of your medication is through a medicine take-back program. Talk to your pharmacist or contact your local garbage/recycling department to learn about take-back programs in your community. See the FDA's Safe Disposal of Medicines website ( for more information if you do not have access to a take-back program.

Side effects

The most common side effects associated with Benclamet are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Appropriate studies have not been performed on the relationship of age to the effects of Benclamet in the pediatric population. Safety and efficacy have not been established.

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Mycotic pseudoaneurysm presenting as a pulsatile mass at sternotomy incision. Eighty percent of patients experienced dramatic improvement in symptoms that had a lasting effect after the initial treatment. The effect of albumin, globulin, pus and dead bacteria in aerobic and anaerobic conditions on the antibacterial activity of moxifloxacin, trovafloxacin and ciprofloxacin against Streptococcus pneumoniae, Staphylococcus aureus and Escherichia coli. Racial differences in blood pressure magnitude begin earlier in life in African-Americans, and vascular damage and target organ injury occurs more rapidly. At 1-year of follow-up after hepatic R0 resection/RFA, there was no evidence of tumor recurrence or extrahepatic metastasis. The aim of the present study was to investigate three weeks of octreotide and captopril treatment alone or in combination following three months of untreated experimental diabetes, and compare the effects to those bactrim dose pediatric mrsa of insulin treatment. Relapse carries a high mortality risk and none of the patients with LR eventually survived. Furthermore, only the net effect of IL-1 was inhibited by IL-1ra, even when human neutrophil was stimulated with IL-1 plus other cytokines to release O2-. However, all these effects were much more pronounced in cells stably transduced with the dominant negative-Arg-175His mutant p53 (DN-R175H mutant p53), which also showed loss of epithelial cytoarchitecture and extensive E-cadherin downregulation. However, our observations that TAP complexes containing two identical TAP NBDs can mediate translocation indicate that distinct properties of the nucleotide binding site per se are not essential for the TAP catalytic cycle.

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Available H-reflex procedures do not allow a reliable or valid examination for the L5 radiculopathy. One is the induction of exon skipping to correct out-of-frame to in-frame dystrophin mRNA, thereby leading to the production of truncated dystrophin. Moreover, a comprehensive metabolomics analysis revealed that the levels of glucose 6-phosphate and 6-phosphogluconate as early metabolites in glycolysis and PPP were decreased after inhibition of the PI3K/AKT/mTOR pathway, suggesting a link between PI3K signaling and the proper function of glucose transporters or hexokinases in glycolysis. Using thermal annealing, we completed the synthesis of the triacetylene polymer. Molecular characterisation of 34 patients with biotinidase deficiency ascertained by newborn screening and family investigation. Respiratory pauses were produced using conscious sedation and positive-pressure face-mask ventilation. Specifically during unconscious face processing, the PTSD, compared with all other groups, showed higher right basolateral (BLA) amygdala activity to fearful v. Multiple-probe thermography for estimating the postmortem interval: I. In addition, HR reduction by pharmacological intervention with ivabradine (a selective HR-lowering agent that acts by inhibiting the pacemaker ionic current I(f) in sinoatrial node cells) reduces the formation of atherosclerotic plaques in animal models of lipid-induced atherosclerosis.

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